← All chapters

Biliary Disease

Gallstones and their complications — from asymptomatic stones through cholecystitis, ductal obstruction, cholangitis, and biliary malignancy.

High-Yield Points

  • Cholesterol stones (most common in the US) form from stasis + decreased bile salts/lecithin; pigment stones are most common worldwide — black stones from hemolysis/cirrhosis/TPN (in the gallbladder), brown stones from biliary stasis/infection (primary CBD stones, often E. coli β-glucuronidase).
  • Biliary colic = transient cystic-duct obstruction, pain <4–6 h, normal labs/US inflammationelective laparoscopic cholecystectomy.
  • Acute cholecystitis = cystic-duct obstruction + inflammation: fever, positive Murphy sign, wall ≥4 mm/pericholecystic fluid on US. RUQ ultrasound is the best initial test; HIDA (non-visualization of the gallbladder) when US is equivocal. Treat with IV fluids + antibiotics + early (same-admission) lap chole.
  • Acalculous cholecystitis — critically ill/TPN/burns/trauma, no stonespercutaneous cholecystostomy if unstable. Emphysematous (gas in wall, diabetic men) → Clostridium perfringens → emergent cholecystectomy.
  • Choledocholithiasis: jaundice + dilated CBD (>6–8 mm) + ↑alk phos/bilirubin. High probability/visualized stone → ERCP; intermediate probability → MRCP/EUS/IOC first.
  • Ascending cholangitis = obstructed, infected biliary tree. Antibiotics + IV fluids FIRST, then ERCP decompression (PTC if ERCP fails). E. coli is the #1 organism.
  • Gallstone ileus — large stone erodes a cholecystoduodenal fistula, lodges at the terminal ileum; Rigler triad (SBO + pneumobilia + ectopic stone) → enterolithotomy, leave the fistula.
  • Biliary dyskinesia — biliary pain, no stones, CCK-HIDA ejection fraction <35–40% → laparoscopic cholecystectomy.
  • Gallbladder adenocarcinoma (most common biliary-tract cancer) — risk = gallstones, large (>3 cm) stones, porcelain GB; Tis/T1 → simple cholecystectomy; T2–T4 → radical cholecystectomy (segments IVb/V + portal nodes). Porcelain gallbladder → cholecystectomy.
  • Cholangiocarcinomapainless jaundice, weight loss, ↑bili/alk phos; risk = PSC/UC, choledochal cysts, liver flukes. MRCP to map, ERCP brushings for tissue, CA 19-9. Klatskin (hilar) = most common, worst prognosis.
  • Post-cholecystectomy bile leak = usually cystic-duct stump → US → drain biloma → ERCP sphincterotomy ± stent. Complete CBD transection (Strasberg E) → Roux-en-Y hepaticojejunostomy (primary repair strictures due to 3- and 9-o'clock blood supply).

Key Tables / Differentials

SyndromePainFever/WBCLFTsUS/KeyFirst step
Biliary colic<4–6 h, resolvesNoNormalStones, no wall changeElective lap chole
Acute cholecystitisConstant >6 h, Murphy+YesMild ↑Wall ≥4 mm, fluidUS → abx + early lap chole
CholedocholithiasisRUQ, jaundiceNo↑↑ alk phos/biliDilated CBD ± stoneERCP (or MRCP if intermediate)
CholangitisCharcot triadYes↑↑Dilated CBDAbx + fluids → ERCP

Board Pearls

  • Charcot triad (RUQ pain + fever + jaundice) → cholangitis; + shock + AMS = Reynolds pentad → emergent ERCP decompression.
  • Air in the biliary tree (pneumobilia) → most commonly prior ERCP/sphincterotomy; with SBO → gallstone ileus.
  • Highest PPV for biliary obstruction = alkaline phosphatase.
  • Calot's (hepatocystic) triangle = cystic duct / common hepatic duct / liver edge; cystic artery = branch of the right hepatic artery; confirm the critical view of safety (only 2 structures) before clipping.
  • Mirizzi syndrome — stone in the GB neck/cystic duct compressing the common hepatic duct.

Classic Vignette Triggers

  • "Gas in the gallbladder wall in a diabetic" → emphysematous cholecystitis (Clostridium).
  • "ICU patient on TPN, RUQ pain, no stones" → acalculous cholecystitis → cholecystostomy.
  • "Elderly woman, SBO, pneumobilia, ectopic stone" → gallstone ileus → enterolithotomy.
  • "Painless jaundice + ulcerative colitis + hilar stricture" → cholangiocarcinoma (Klatskin).
  • "POD#3 after lap chole, bilious drain/biloma" → cystic-duct stump leak → ERCP + stent.